A 60 year old male who is a known patient with type 2 diabetic mellitus and hypertension presents to your clinic and has the below ECG:
What are the ECG abnormalities?
What is the differential diagnosis of this ECG finding?
A 21 year old healthy female comes to the clinic with complaints of intermittent palpitations. These episodes can last from seconds to minutes and are associated with lightheadedness, diaphoresis, and occassionally chest pain. Her ECG is below:
What is the Abnormality in ECG 1?
What is the Abnormality in ECG 2?
An 87 year old male with a history of coronary artery disease has the below ECG:
ECG ANSWER
1) Normal sinus rhythm with first degree AV block
2) Right bundle branch block (RBBB)
3) Left anterior fascicular block (LAFB or left anterior hemiblock or LAHB)
4) PVC
A 58 year old male with a history of DM, hypertension, and dyslipidemia presents with substernal chest pressure for 30 minutes. The pain began at rest,severe in nature, was mild to begin with then increased in intensity, is worse with exertion, radiates to his left arm, is not pleuritic or positional, and is associated with diaphoresis, shortness of breath, and nausea.
His temperature is 37.2, blood pressure 90/50mmhg, heart rate 100/bpm, respiratory rate is 16/min. No jugular venous distension is present, he is tachycardic and an S4 heart sound is present. No murmurs are heard. His lungs are clear to auscultation. The rest of his exam is normal.
ECG ANSWER
1) Normal sinus rhythm
2) Poor R wave progression (possibly indicated an anterior wall infarct - age undetermined)
3) Old inferior wall infarct (indicated by Q waves in inferior leads)
4) Biphasic T waves in leads V1 - V4 possibly consistent with ischemia
A 72 year old male receives a routine ECG which is below. His electrolytes are normal.
ECG ANSWER
The PR interval in this ECG is very long, about 380 milliseconds (normal is 120 - 200 ms or 0.12 - 0.20 seconds). A P wave preceeds each QRS complex, thus this is 1st degree AV nodal block.
An 82 year old male with a history of hypertension presents to the clinic with a complaint of generalized weakness for 3 days. No chest pain, shortness of breath, dyspnea on exertion, lower extremity edema, paroxysmal nocturnal dyspnea, orthopnea, palpitations, or dizziness. He takes lisinopril and hydrochlorothiazide for his hypertension.
His temperature is 37.0, blood pressure 100/50 mmHg, heart rate 155 bpm, respiratory rate is 16/min. No jugular venous distension is present, he is tachycardic and irregularly irregular on cardiac exam without any murmurs. His lungs are clear to auscultation. The rest of his exam is normal.
His ECG is below
ECG ANSWER
The ECG findings include:
1) Atrial fibrillation with rapid ventricular response
2) PVC(Premature ventricular contraction)
Atrial fibrillation occurs when the atrial conduction becomes chaotic and very fast. The atrial rate increases to around 400-600 beats per minute. When this occurs, the amplitude of the P waves markedly decrease. Frequently, the P waves are not able to be detected at all, however at times a coarseness of the baseline of the ECG occurs reflecting the fibrillatory atrial activity. Not all of the 400-600 atrial action potentials per minute are conducted to the ventricles otherwise ventricular fibrillation would occur. Instead, the AV node is able to block a good number of beats usually resulting in a ventricular rate between 120-180 in the absence of AV blocking medications. Due to the chaotic atrial activity, varying block of the atrial action potentials at the AV node occurs resulting in an irregularly irregular rhythm.
A 68 year old male with a past history of chronic atrial fibrillation presents to the emergency room with a complaint of nausea, vomiting, generalized abdominal pain, and generalized weakness. No fevers or chills. No diarrhea, constipation, melena, or other signs of gastrointestinal bleeding. He does not know his medications. He states his vision has been slightly yellow tinged recently.
His temperature is 37.0, blood pressure 90/50, heart rate 55, respiratory rate is 16. No jugular venous distension is present, he is bradycardic and irregularly irregular on cardiac exam without any murmurs. His lungs are clear to auscultation. His abdominal exam is completely normal. The rest of his exam is normal.
His ECG is below:
ECG ANSWER
1) atrial fibrillation with bradycardia
2) ST segment depression consistent with digoxin effect
The ECG shows a downsloaping ST segment depression in multiple leads that are shaped like the "reverse check mark sign" or a "scooped out" appearance. This pattern is typical of digoxin. This may occur at normal digoxin levels in some people, but becomes more common as the digoxin level increases.
Her temperature is 37.0, blood pressure 90/50, heart rate 190, respiratory rate is 16. No jugular venous distension is present, she is tachycardic and regular on cardiac exam without any murmurs. Her lungs are clear to auscultation. The rest of her exam is normal.
Identify this ECG rhythm?
ECG ANSWER
Narrow complex tachycardia - most likely AV nodal rentry tachycardiaa (AVNRT)
The ECG shows a narrow-complex tachycardia that is regular.No flutter waves are seen. In leads V3 - V6 a P wave can be seen just after the QRS complex making this rhythm a "short-PR" tachycardia. Remember the most common short-PR tachycardia is AV nodal rentry tachycardia.
A 86 year old male with no significant past medical history to clinic with a complaint of generlized weakness, dizziness, and dyspnea on exertion. Mild lower extremity edema, paroxysmal nocturnal dyspnea, and orthopnea are present. No chest pain has occured. He is not taking any medications.
His temperature is 37.0, blood pressure 80/40mmHg, heart rate 40/BPM, respiratory rate is 16/min. Mild jugular venous distension is present, he is bradycardic and regular without any murmurs. His lungs are clear to auscultation. The rest of his exam is normal.
His ECG is below:
ECG ANSWER
A junctional rhythm occurs when the SA node is not able to generate an action potential and AV node acts as the main pacemaker of the heart. In this instance, the P wave morphology (normally generated when the SA node fires causing atrial depolarizaion) will be altered since its origin is no longer at the SA node and is instead at the AV node. The direction of conduction to the atria in a junctional rhythm is considered "retrograde" since it is traveling up the atria from the AV node instead of down the atria from the SA node. This results in an inverted P wave in lead II (normally upright in sinus rhythm) and an upright P wave in aVR (normally inverted in sinus rhythm).
Depending on the exact location of the pacemaker in a junctional rhythm, the P wave location may vary. If it occurs slightly above the AV node (low atrium), then the P wave will actually occur slightly before the QRS complex is seen. This will result in the P wave appearing just before the QRS, but PR interval will be short (< 0.12 ms). If the pacemaker occurs at the junction, the P wave and QRS complex may be exactly overlying and you may not be able to identify any P waves at all. If the pacemaker occurs lower in the junction, the QRS complex may actually occur before the P wave (the P wave will be seen after the QRS complex). In this case a retrograde P wave can be identified just before the QRS complex with a short PR interval, thus the pacemaker is located high in the AV node or perhaps in the low atrium. Causes of a junction rhythm include chronic degeneration of the SA node (termed "sick sinus syndrome"), hypoxia, myocardial ischemia, hyperkalemia, digoxin toxicity, elevated vagal tone, and medications known to supress the SA node (beta-blockers and non-dihydropyradine calcium channel blockers).
A 50 year old male with no past medical history presents to the emergency room with sudden onset severe chest pain. The pain is 10/10, substernal, sharp in nature, worse with deep inspiration, not positional or reproducible, and associated with shortness of breath and dizziness but no excessive sweating , nausea, or vomiting.
His temperature is 37.0, blood pressure 80/40, heart rate 110, respiratory rate is 28. His oxygen saturation is 88% on room air. He appears in mild distress, no jugular venous distension is present, he is tachycardic and regular without any murmurs. His lungs are clear to auscultation. The rest of his exam is normal.
His ECG is given below:
ECG ANSWER
1) Sinus tachycardia
2) S1Q3T3 pattern consistent with acute cor pulmonale (acute right ventricular strain) a.k.a. the "McGinn-White sign"
The diagnosis is pulmonary embolism
The patient's clinic scenario of pleuritic chest pain, tachycardia, and hypoxia should raise a high clinical suspicion for pulmonary embolus. Also, his chest pain was sudden onset and severe unlike that of myocardial ischemia. Aortic disection can cause sudden-onset severe chest pain as well, however usually the patient is hypertensive and not necessarily hypoxic.
A 53 year old male with a history of uncontrolled hypertension is found unresponsive at the side of the street and is brought to the emergency room.
His temperature is 37.0, blood pressure 190/100, heart rate 50, respiratory rate is 8. He is unrepsonsive on physical exam. Heart and lung exams are normal. Laboratory studies including electrolytes and troponin levels are all normal.
His ECG is below:
ECG ANSWER
1) Normal sinus rhythm
2) Marked T wave inversions and prolonged QT interval
Answer: A CT scan of the brain is needed. His clinical picture and ECG findings are consistent with an acute central nervous system abnormality. Patients with subarachnoid hemorrhages, intracranial hemorrhages, and less commonly severe ischemic strokes have ECGs that reveal deep, symmetric T wave inversions and QT prolongation. Less commonly, carotid endarterectomy can result in this ECG pattern chronically.
A 27 year old female with a history of HIV infection presents to the emergency room with a complaint of increased dyspnea on exertion and lower extremity edema over the past 6 months. She denies chest pain, paroxysmal nocturnal dyspnea or orthopnea.
Her temperature is 37.0, blood pressure 90/50, heart rate 110, respiratory rate 20. She has large V waves in her jugular venous pulsations, a III/VI pansystolic murmur at the left lower sternal border which gets louder with inspiration, and a right ventricular S4 heart sound is heard. A pulsatile liver is palpated.
Her ECG and chest x-ray are given below:
ECG ANSWER
1) Sinus tachycardia
2) RVH with strain pattern
Chest XRAY ANSWER
A 23 year old female medical student presents to the emergency room with altered mental status. Her friends say she has been drinking hard liquor non-stop for 1 week after her final exams and has not been eating well for months. She was recently started on erythromycin for a upper respiratory tract infection.
Her temperature is 37.0, blood pressure 90/50, heart rate 70, respiratory rate 10. She is thin and cachectic appearing. She is arousable but unable to answer questions or follow commands. Her heart and lung exam are normal.
Her laboratory studies reveal a potassium level of 2.1 (normal 3.5-5.0), a magnesium level of 0.9 (normal 1.8-3.0), and a calcium level of 5.0 (normal 9.0-10.5). Her ECG is below:
Identify the abnormalities in 1st and 2nd ECG strips?
ECG 01 ANSWER
1) Normal sinus rhythm
2) Prolonged QT interval
She was pre-disposed to a long QT due to hypokalemia, hypomagnesemia, hypocalcemia, erythromycin use and female gender.












